Hydrocortisone
Introduction
- synthetic cortisol
- cannot be bioanalytically separated from endogenous cortisol [๐]
- the relatively short half-life of 1.5 h
- lack of licensed formulation for this population (for <6yo)
- Dose
- modified release formulations [๐]
- Plenadren
- Chronocort
- administration
- subcutaneously via insulin -> circadian cortisol profile and improved 17-OHP concentrations [๐]
- BCS (biopharmaceutical classification system) class II [๐]
- low dissolution rate and high intestinal permeability [๐]
Pharmacokinetics
Absorption
- Parameters
- passive diffusion and is a poor substrate for the efflux protein P-glycoprotein [๐]
- \(T_{max}\)
- tablet 1-1.7 h
- note: 1.7 h in high dose 50 mg [TBA]
- solution 0.7-1 h
- pediatric adrenal insufficiency 0.33-3 h
- tablet 1-1.7 h
- \(t_{1/2,abs}\) [๐]
- fasting: 15 min
- fed: 43 min
- \(C_{max}\)
- 75th percentile of observed data [๐]
- F
- 100% when dose of <20 mg
- 54% when dose of 50 mg in patients with adrenal insufficiency [๐]
Distribution
- Parameters
Metabolism
- [๐]
- 11ฮฒ-hydroxysteroid dehydrogenase
- bidirectional
- type 1 (11ฮฒ-HSD1) to cortisol liver and adipose
- undetectable \(<3\) months
- stabilized approx. at 12 months
- type 2 (11-ฮฒHSD2) to cortisone in kidney
- increased from 0-52 weeks,
- 5ฮฑ-reductase(cortisol) and 5ฮฒ-reductase (cortisone)
- unidirectional
- low in newborns
- highest activity >3 months
- declined <52 weeks
- similar 3.5-17.5 years
- unidirectional
- 3ฮฑ-hydroxysteroid
- unidirectional
- 6ฮฒ-hydroxylase (CYP3A4)
- unidirectional
- approx. 1%
- 11ฮฒ-hydroxysteroid dehydrogenase
Metabolic pathway of cortisol. Figures were adapted from PhD thesis of Johanna Melin. Created using diagrams.net
Elimination
- At least 90% of cortisol and cortisone metabolites are normally excreted as sulfate or glucuronide conjugates. Conjugation of cortisol and cortisone can occur at the 21 position, and 3ฮฑ-reduced steroids can also be conjugated at the 3ฮฑ position
- Parameters
- \(CL\)
- dose-dependent CL
- lower in neonates (due to maturation of metabolic enzymes)
- 12.5 to 20.2 L/h (iv 5-50 mg)
- 12.5 L/h (iv 5 mg)
- 17.6 L/h (iv 40 mg)
- note: Saturation of CBG leads to \(CL_u\) increasing disproportionally with respect to \(CL_{tot}\).
- CL (\(CL_u\)) remains constant, since it is independent of \(f_u\).
- \(CL/F\)
- 19.1 L/h (oral 10-20 mg: Fโ1)
- 27.3 L/h (50 mg)
- 12.4-25.6 L/h (patients with adrenal insufficiency)
- note: highest value in pubertal patients
- \(AUC\)
- nonlinear processes (e.g. saturable plasma protein binding or saturable absorption)
- \(CL\)
Pharmacodynamics
- Effect
- endogenous cortisol synthesis
- T cell dynamics [๐]
- correlation with 17-OHP concentration
- 1 h post dose
- delayed inhibitory effect with r=-0.302 [๐]
- no correlation between AUC for cortisol and 17-OHP [๐]
Formulations and Administration
- Lowest available dose for a oral tablet
- 5 mg in USA
- 10 mg in Europe or Canada
Infacort ยฎ
- Diurnal Ltd
- immediate-release hydrocortisone
- Brand name: Alkindi (with four doses: 0.5 mg, 1 mg, 2 mg and 5 mg)
- Approval for children with adrenal insufficiency from regulatory agencies [๐]
- EMA - 2018
- FDA - 2020
- oral (po)
- hydrocortisone granules with taste masking
- dry granules or mixed with a small amount of soft food
- analysis in adults with a NCA by [๐]
- does not account for known nonlinearities of cortisol
- good disease control with normal growth and no increased risk of adrenal crises [๐]
Licensed Hydrocortisone tablet
- Auden Mackenzie Ltd
- oral (po)
Hydrocortisone Succinate
- intravenous (iv) bolus
- HC administration
Hydrocortisone cypionate suspension
- Pharmacia & UpJohn
- voluntarily recalled in July 2000 [๐]
- possible inaccuracies in preparations can lead to unanticipated adverse effects [๐]